Botulism &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; &nbs 626j91g p; 165
General: Caused by a toxin-producing strain of Clostridium botulinum; occurs primarily after the ingestion of contaminated food; the organism can produce a neurotoxin, the effect of which can be life threatening.
Ocular: Absent optokinetic nystagmus, absent vertical gaze; marked limitation of horizontal gaze; ptosis; diplopia; decreased tear secretion; mydriasis; paralysis of accommodation; nystagmus; optic atrophy; optic neuritis; extraocular muscle paresis.
Clinical: Dizziness; severe respiratory impairment; gastrointestinal disturbances; dysphagia; dysarthria; postural hypotension.
Fraunfelder FT, Roy FH. Current Ocular Therapy, 5th ed. Philadelphia: WB Saunders, 2000.
Hambleton P. Clostridium botulinum toxins: a general review of involvement in disease, structure, mode of action and preparation of Clinical use. J Neurol 1992; 239:16.
Hedges TR, et al. Botulin ophthalmoplegia. Arch Ophthalmol 1983; 101:211-213.
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